Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
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Related Images of Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
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Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
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Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
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Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
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Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
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Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
Lis1 Interacts With Clip170 To Promote Tumor Growth And Metastasis Via
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Physical Interaction Between Lis1 And Clip 170 A Lis1 Interacts With
Physical Interaction Between Lis1 And Clip 170 A Lis1 Interacts With
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Pdf Lis1 Interacts With Clip170 To Promote Tumor Growth And
Pdf Lis1 Interacts With Clip170 To Promote Tumor Growth And
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A Model Of The Possible Interactions Between Clip 170 Open I
A Model Of The Possible Interactions Between Clip 170 Open I
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Figure 1 From Arsenic Trioxide Disturbs The Lis1ndel1dynein
Figure 1 From Arsenic Trioxide Disturbs The Lis1ndel1dynein
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Lis1 Clip 170s Key To The Dyneindynactin Pathway Abstract
Lis1 Clip 170s Key To The Dyneindynactin Pathway Abstract
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Lis1 Clip 170s Key To The Dyneindynactin Pathway Abstract
Lis1 Clip 170s Key To The Dyneindynactin Pathway Abstract
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Lis1 And P150glued Compete For Binding To The Cooh Terminus Of
Lis1 And P150glued Compete For Binding To The Cooh Terminus Of
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Dynein Lis1 And Clip‐170 Counteract Eg5‐dependent Centrosome
Dynein Lis1 And Clip‐170 Counteract Eg5‐dependent Centrosome
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Lis1 Clip 170s Key To The Dyneindynactin Pathway Abstract
Lis1 Clip 170s Key To The Dyneindynactin Pathway Abstract
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Figure 2 From Arsenic Trioxide Disturbs The Lis1ndel1dynein
Figure 2 From Arsenic Trioxide Disturbs The Lis1ndel1dynein
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Structure Of Lis1 Binding To Dynein At Sitestalk A An Overlay Of
Structure Of Lis1 Binding To Dynein At Sitestalk A An Overlay Of
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Effect Of P50 Overexpression On Clip 170 And Lis1 Targeting To
Effect Of P50 Overexpression On Clip 170 And Lis1 Targeting To
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Figure 6 From Arsenic Trioxide Disturbs The Lis1ndel1dynein
Figure 6 From Arsenic Trioxide Disturbs The Lis1ndel1dynein
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Dynein Lis1 And Clip‐170 Counteract Eg5‐dependent Centrosome
Dynein Lis1 And Clip‐170 Counteract Eg5‐dependent Centrosome
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Equilibrium Binding Model For Vertebrate Eb1 And Clip 170 Plus End
Equilibrium Binding Model For Vertebrate Eb1 And Clip 170 Plus End
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The Cooh Terminus Of Clip 170 Interacts With The Nh2 Termini Of Both
The Cooh Terminus Of Clip 170 Interacts With The Nh2 Termini Of Both
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Lis1 Regulates The Association Of Dynein And Dynactin Components A
Lis1 Regulates The Association Of Dynein And Dynactin Components A
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Msi1 Interacts With Ago2 In The Cytosol And Promote Tumor Growth A
Msi1 Interacts With Ago2 In The Cytosol And Promote Tumor Growth A
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Ros Mediated Signaling In Promotion Of Breast Tumor Growth Metastasis
Ros Mediated Signaling In Promotion Of Breast Tumor Growth Metastasis
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Emdscs Promote Tumor Growth Via Accelerating Emt Process A
Emdscs Promote Tumor Growth Via Accelerating Emt Process A
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Lis1 Interacts With Numa1 And Regulates Ais Structure A Div7
Lis1 Interacts With Numa1 And Regulates Ais Structure A Div7
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Primary Tumors Release Itgbl1 Rich Extracellular Vesicles To Promote
Primary Tumors Release Itgbl1 Rich Extracellular Vesicles To Promote
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Mlk7 As1 Interacted With Yap1 To Promote Tumor Growth Metastasis And
Mlk7 As1 Interacted With Yap1 To Promote Tumor Growth Metastasis And
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Pro Inflammatory Tnf α And Ifn γ Promote Tumor Growth And Metastasis
Pro Inflammatory Tnf α And Ifn γ Promote Tumor Growth And Metastasis
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The Vicious Cycle Induced By Gal 8 To Promote Tumor Growth And
The Vicious Cycle Induced By Gal 8 To Promote Tumor Growth And
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Structure Of Lis1 Binding To Dynein At Sitering A Lis1 At Sitering
Structure Of Lis1 Binding To Dynein At Sitering A Lis1 At Sitering
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Pdf Mait Cells Promote Tumor Initiation Growth And Metastases Via
Pdf Mait Cells Promote Tumor Initiation Growth And Metastases Via
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Lis1 Acts As A Clutch” Between The Atpase And Microtubule Binding
Lis1 Acts As A Clutch” Between The Atpase And Microtubule Binding
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Lis1 And Mecp2 Interaction A Nuclear Localization Of Lis1 Hela
Lis1 And Mecp2 Interaction A Nuclear Localization Of Lis1 Hela
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