Rev7 And Rev3l Protect Activated Ch12 Cells From Cell Death A Survival
Rev7 And Rev3l Protect Activated Ch12 Cells From Cell Death A Survival
Rev7 And Rev3l Protect Activated Ch12 Cells From Cell Death A Survival
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Rev7 Interacts With Full Length Rev3l In Vivo A Schematic Of Rev3l
Rev7 Interacts With Full Length Rev3l In Vivo A Schematic Of Rev3l
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Rev7 Interacts With Full Length Rev3l In Vivo A Schematic Of Rev3l
Rev7 Interacts With Full Length Rev3l In Vivo A Schematic Of Rev3l
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Both Rev7 Binding Sites In Rev3l Are Important For Cellular Resistance
Both Rev7 Binding Sites In Rev3l Are Important For Cellular Resistance
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Rev7 Interacts With Full Length Rev3l In Vivo A Schematic Of Rev3l
Rev7 Interacts With Full Length Rev3l In Vivo A Schematic Of Rev3l
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Rev7 Directs Dna Repair Pathway Choice Trends In Cell Biology
Rev7 Directs Dna Repair Pathway Choice Trends In Cell Biology
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Rev3l Is Essential To Protect And Restart Stalled Replication Forks A
Rev3l Is Essential To Protect And Restart Stalled Replication Forks A
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Expression Of Human Rev3l Complements Rev3l Deficient Mefs A Top
Expression Of Human Rev3l Complements Rev3l Deficient Mefs A Top
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Rev7 Is Required For Processing Aid Initiated Dna Lesions In Activated
Rev7 Is Required For Processing Aid Initiated Dna Lesions In Activated
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Model Employing For Promotion Of Functional Interactions Of Rev7 By Two
Model Employing For Promotion Of Functional Interactions Of Rev7 By Two
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Apim Mediated Rev3l⁻pcna Interaction Important For Error Free Tls Over
Apim Mediated Rev3l⁻pcna Interaction Important For Error Free Tls Over
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Increased Expression Of Rev7 In Small Cell Lung Carcinomas And Its
Increased Expression Of Rev7 In Small Cell Lung Carcinomas And Its
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A Novel Rev7 Binding Site In Rev3l A In Vitro Association Of Rev7
A Novel Rev7 Binding Site In Rev3l A In Vitro Association Of Rev7
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Crystal Structure Of Human Rev7 In Complex With A Human Rev3 Fragment
Crystal Structure Of Human Rev7 In Complex With A Human Rev3 Fragment
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Knock In Mice And Mefs Expressing Active Site Mutant Rev3l Have
Knock In Mice And Mefs Expressing Active Site Mutant Rev3l Have
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Rev7 Is Essential For Dna Damage Tolerance Via Two Rev3l Binding Sites
Rev7 Is Essential For Dna Damage Tolerance Via Two Rev3l Binding Sites
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Biological Roles Of Rev7 A Crystal Structure Of The Complex Between
Biological Roles Of Rev7 A Crystal Structure Of The Complex Between
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Rev7 Protein Expression And Csr In Complemented Rev7−− Ch12 F3
Rev7 Protein Expression And Csr In Complemented Rev7−− Ch12 F3
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Rev3l The Catalytic Subunit Of Dna Polymerase ζ Is Involved In The
Rev3l The Catalytic Subunit Of Dna Polymerase ζ Is Involved In The
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P31 Inhibits Rev7 Activity In Icl Repair A Graph Showing The
P31 Inhibits Rev7 Activity In Icl Repair A Graph Showing The
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Rev3l Expression Is Important For Cellular Resistance To Cisplatin In
Rev3l Expression Is Important For Cellular Resistance To Cisplatin In
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Mutational Screening Reveals Rev7 Interaction Surfaces Essential For
Mutational Screening Reveals Rev7 Interaction Surfaces Essential For
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Rapid Senescence And Apoptosis After Deletion Of Rev3l A Cell Cycle
Rapid Senescence And Apoptosis After Deletion Of Rev3l A Cell Cycle
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Effect Of Rev7 Sirna On Phosphoinositide 3 Kinase Pi3kakt Signaling
Effect Of Rev7 Sirna On Phosphoinositide 3 Kinase Pi3kakt Signaling
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Rev7 Is Required For Processing Aid Initiated Dna Lesions In Activated
Rev7 Is Required For Processing Aid Initiated Dna Lesions In Activated
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Rev3l Knockdown Inhibits Escc Cell Proliferation And Invasion A Te 1
Rev3l Knockdown Inhibits Escc Cell Proliferation And Invasion A Te 1
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9 Full Length Human Rev3l Does Not Interact With Rev7 Irrespective Of
9 Full Length Human Rev3l Does Not Interact With Rev7 Irrespective Of
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Influence Of Upregulation Or Downregulation Of Rev3l On Rev7l
Influence Of Upregulation Or Downregulation Of Rev3l On Rev7l
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Overexpression Of Rev7 In Glioma Tissue A B The Expression Of Rev3l
Overexpression Of Rev7 In Glioma Tissue A B The Expression Of Rev3l
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Rev3l Knockdown Can Efficiently Synergize The Apoptosis Response To
Rev3l Knockdown Can Efficiently Synergize The Apoptosis Response To
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Champ1 Binds To Rev7fancv And Promotes Homologous Recombination Repair
Champ1 Binds To Rev7fancv And Promotes Homologous Recombination Repair
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Rev3l Is Negatively Regulated By Mir 29a In Non Small Cell Lung Cancer
Rev3l Is Negatively Regulated By Mir 29a In Non Small Cell Lung Cancer
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